Researchers identified a potential new way of fighting HIV infection that relies on the remnants of ancient viruses, human endogenous retroviruses (HERV).
Mounting evidence suggests that HIV infection could enable HERV expression by disrupting the normal controls that keep HERV in check. In some HIV-infected individuals this induces infection fighting T cells to target HERV expressing cells.
The scientists looked at 29 individuals recently infected with HIV from the University of California San Francisco OPTIONS Project and 13 HIV-negative individuals and 3 hepatitis C infected, HIV-negative individuals from Toronto. In the group recently infected with HIV, researchers found a relationship between increased T-cell response to HERV and decreased levels of HIV virus present in patients blood.
Genetic Engineering & Biotechnology News
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